AND ALZHEIMER'S DISEASE.
Scientists from Lund University in Sweden (Lund University) discovered a new property of vitamin C: it dissolves the toxic protein accumulations that form in the brain in Alzheimer's disease. The results of the study were published by experts in the Journal of Biological Chemistry.
As the authors explain, people suffering from Alzheimer's disease produce an increased amount of beta-amyloid peptides, which accumulate in the form of plaques in certain parts of the brain. They cause the death of nerve cells. First of all, they suffer from neurons located in the brain areas responsible for memory.
"When we treated the brain tissue of sick mice with vitamin C, we found that toxic protein deposits dissolved. Our research revealed a previously unknown function of the vitamin, we showed its ability to affect amyloid plaques," says molecular medicine specialist Katrin Mani. She also noted that vitamin C does not have to be ingested from fresh fruit. "Experiments have shown that vitamin C can be obtained in large quantities in the form of dehydroascorbic acid from juice, which, for example, stood in the refrigerator all night," says K. Mani.
At the moment, there is no valid method to combat Alzheimer's disease. There are only means to mitigate the symptoms and inhibit the development of the disease. An antioxidant such as vitamin C, which has a protective effect on many diseases (from the common cold to heart attacks and dementia), has long been the center of attention of scientists. According to K. Mani, it is unambiguous to assert that vitamin C has a positive effect on the disease, it is too early, but the obtained results open up new possibilities for the study of the disease and the therapeutic properties of vitamin C.
Earlier, scientists from the Mount Sinai School of Medicine in New York City concluded that grape seeds are helping in the fight against Alzheimer's disease. This is explained by the fact that they are rich in polyphenols - a type of antioxidants that protects cells and body chemicals from damage caused by free radicals. In turn, their colleagues from the Buck Institute for Age Research (Buck Institute for Age Research) stated that the progression of the disease can slow lithium, as it prevents the formation of amyloid plaques and neurofibrillary tangles in brain cells.